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dc.contributor.authorNguyen, AD
dc.contributor.authorSlack, K
dc.contributor.authorSchwarzer, C
dc.contributor.authorLee, NJ
dc.contributor.authorBoey, D
dc.contributor.authorMacia, L
dc.contributor.authorYulyaningsih, E
dc.contributor.authorEnriquez, RF
dc.contributor.authorZhang, L
dc.contributor.authorLin, S
dc.contributor.authorShi, YC
dc.contributor.authorBaldock, PA
dc.contributor.authorHerzog, H
dc.contributor.authorSainsbury, Amanda
dc.date.accessioned2017-01-24
dc.date.available2017-01-24
dc.date.issued2014-06-01
dc.identifier.urihttp://hdl.handle.net/2123/16245
dc.publisherNeuropeptidesen_AU
dc.subjectobesityen_AU
dc.subjectneuropeptide Yen_AU
dc.subjectendocrinologyen_AU
dc.subjectenergy metabolismen_AU
dc.subjectcentral feeding regulationen_AU
dc.titleDouble deletion of orexigenic neuropeptide Y and dynorphin results in paradoxical obesity in miceen_AU
dc.typeArticleen_AU
dc.identifier.doi10.1016/j.npep.2014.03.001
dc.type.pubtypePost-printen_AU


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